Adipo-X™ Advanced Fat-Loss Technology - Lose Weight Fast!
Adipo-X™ is the most effective, most innovative, scientifically engineered fat
loss formula designed to boost your metabolism, heighten energy levels and improve
mental focus and vigor. It uses a liquid capsule delivery system for rapid maximum
absorption. Adipo-X™ increases the metabolism safely and naturally with the most
unique fat burning formula ever developed. Adipo-X™ destroys fat and lets you get
ripped, while allowing you to retain your hard earned muscle.
The key to Adipo-X™’s successful fat loss technology is a multiple pathway approach.
Unlike many other weight loss products that try to tackle fat loss through one biochemical
pathway or ingredient, Adipo-X™ approaches fat loss in seven different ways.
1. Increases the release of the main adrenal fat loss hormones epinephrine and
norepinephrine
2. Enhances the fat loss signal molecule by preventing the messenger molecule cAMP
from being broken down
3. Prevents the fat loss hormones from being broken down, which allows the fat loss
signal to work continuously
4. Prevents alpha adrenergic receptors from stopping the fat loss process
5. Increases the thermogenic uncoupling process
6. Prevents fat synthesis following meals high in sugar or fat
7. Supports thyroid function
The seven step approach to fat loss makes Adipo-X™ the most effective fat burner
developed to date. You haven’t experienced fat loss like this since ephedra. Adipo-X
is the perfect addition to your diet and exercise program and it will:
• Maintain lean body mass while burning fat
• Control Appetite and Prevent overeating
• Ramp up energy metabolism (thermogenic)
• Utilize stored body fat as an energy source (lipolysis)
• Support thyroid function and can help increase insulin sensitivity
• Decrease fatigue
• Improve mental focus, clarity and mood
Detailed overview of the multiple steps Adipo-X™ uses to increase fat loss:
The problem with weight loss products that only approach fat loss through one pathway
or ingredient is that the body has a way of quickly adapting to any attempt to increase
in lipolysis (fat breakdown) or thermogenesis (fat burning). This is called homeostasis,
a method of keeping your body in a specific balanced state. Adipo-X™ uses multiple
pathways to allow your body to enter a state of continual maximal fat burning. Adipo-X™
turns on the fat burning machinery and keeps you in a fat burning state.
1. Increases the release of the main adrenal fat loss hormones epinephrine
and norepinephrine
The ingredients in Adipo-X™ have been scientifically proven to increase the release
of the main fat loss hormones epinephrine and norepinephrine. Taking Adipo-X™ turns
on the first step in fat loss by causing these hormones to be released. These hormones
are known as catecholamines, and they are responsible for the fight or flight (sympathetic)
response. These hormones are primarily responsible for activating fat loss in adipose
tissue through beta adrenergic stimulation.
2. Enhances the fat loss signal molecule by preventing the messenger molecule
cAMP from being broken down
The stimulation of beta adrenergic receptors causes activation of an enzyme that
increases levels of a secondary messenger cyclic AMP (cAMP) in fat cells. Increasing
cAMP causes an increase in lipolysis (fat release) and thermogeneisis (fat burning)
in the fat cells. Cyclic AMP is a very short lived secondary messenger molecule
that is broken down by an enzyme called phosphodiesterase (PDE). Adipo-X™ includes
ingredients that inhibit PDE and cause an increase in cAMP levels within the cells
potentiating the fat loss signal. This allows cAMP to remain active longer, and
it allows the fat loss process to continue working.
3. Prevents the fat loss hormones from being broken down, which allows the fat
loss signal to work continuously
The fat burning response from Adipo-X™ is further enhanced by preventing the breakdown
of the adrenal hormones (norepinephrine and epinephrine). The enzymes COMT (catechol-o-methyltransferase)
and MAO (monoamine oxidase) are the primary enzymes that break down these hormones.
The ingredients in Adipo-X™ have been clinically shown to inhibit the enzymes COMT
and MAO preventing the breakdown of the fat loss hormones. This allow the fat loss
signal to work continuously.
4. Prevents alpha adrenergic receptors from stopping the fat loss process
Activation of the alpha adrenergic receptor slows the fat loss process. The ingredients
in Adipo-X™ prevent activation of the alpha adrenergic receptor, preventing the
fat loss process from slowing down.
5. Increases the thermogenic uncoupling process
The first four steps outlined directly increase the rate of lipolysis (breakdown
of fat) in fat cells. The fifth step is activation of thermogenesis in the body.
Thermogenesis is the actual process of burning the fat once it has been broken down
in the fat cells, and it occurs in the mitochondria of the cell. Thermogenesis turns
the broken down fat into energy that your body can use and body heat.
6. Prevents fat synthesis following meals high in sugar or fat
One unique aspect about Adipo-X™ is that it can prevent fat synthesis following
meals high in sugar or fat. This is done mainly by molecular interactions with the
vanilloid receptor. Adipo-X™ can help keep you on track with your fat loss goals
even if you cheat on your diet and indulge in a meal high in sugar or fat.
7. Supports thyroid function
Adipo-X™ has unique ingredients that support thyroid function. The thyroid is the
main metabolic regulation center. By properly supporting the thyroid the metabolism
is enhanced and fat loss is maximized.
Not taken like any old fat-burner:
How do I take Adipo-X™?
Because Adipo-X™ is a powerful stimulant, we suggest that you start your regimen
with a one capsule serving. After assessing your tolerance of the product, increase
your serving to two capsules before increasing to three. Take each serving of Adipo-X™
with at least eight ounces of water. Never take more than three capsules in a serving
and more than six capsules in a day.
When do I take Adipo-X™?
To complete the thermogenic process, Adipo-X™ contains Capsaicin for its powerful
Heat Units. Because of this, Adipo-X™ must be taken on an empty stomach to avoid
unsettling. Adipo-X™ is best taken on an empty stomach 30 to 60 minutes before eating
breakfast and/or lunch.
It's Time to Begin Your War on Fat!
All of the ingredients in Adipo-X™ work to enhance the body’s ability to burn
fat and this will allow you to get that ripped look you’ve been working so hard
for. Adipo-X™ works so well at increasing metabolism, thermogenesis and lipolysis
that you can actually feel the product working when you take it! A scientifically
designed fat loss formula like this has never been developed before. Forget about
all the other inefficient weight loss products on the market and upgrade to Axis
Labs® Adipo-X™. Get Adipo-X™ now to help you expose your hardest core ever!
Testimonials:
"I tried Adipo-X trying to shed a couple of pounds. It really worked. I saw
results in a week’s time. I have had my daughter try it and she lost 5lbs in the
first week. I, myself, lost 12lbs in two weeks. I feel so much better and my
sex-life is better also. I am not so tired in the middle of sex. My younger
boyfriend even saw the difference. Try it and you will not regret it. This has
been the best diet product that I actually liked and worked for me at the same
time. I walked the treadmill 20min in the morning and 20min in the evening. I
didn't eat at night because I wasn't hungry. I ate fruit and drank plenty of
water. I ate breakfast and a lunch. It really works. I have five of my friends
on them and it is helping them. They ere amazed with the first weeks results. I
give this product 100 stars."
- Lolo, Houston TX
"Great product: I have been using Adipo-X in conjunction with a 1500-1800 kcal
diet, 30 min walk 3 x week and keeping a close eye on my carbs (average 40 per day)
I have lost 7 kgs in 18 days, I diet like this often and usually average around
4 kgs so I put the extra 3 completely in Adipo-X, and I am only taking 3 caps in
the mourning. Love the product and highly recommend it to anyone who is wanting
to lose weight, I am not overweight my BMI is 22 at my biggest so the large weight-lose
isn't due to excess weight but again I believe the extra is completely due to Adipo-X."
- Emma, Auckland, New Zealand
"I have tried all the popular fat loss products over the years, and I can honestly
say that Adipo-X is the only one that delivers. When it comes to my contest physique
and my client's results, I rely on the product that really works, Adipo-X!"
- Jennifer Schumm, Greenwood CO
“Using Adipo-X I have gone from a 40 to a 33 waist”
- G. Williams Athens, GA
Superb! 5 Starts"
- Natalia, Bendery, Moldova
"5 Starts"
- Oxana, Ukraine
Demand more from your fat burner. Demand Axis Labs®.
Supplement Facts
Serving Size: 3 Liquid Capsules
Servings per Container: 40 |
| |
Amount Per Serving |
%DV* |
| Vitamin B1 (Thiamine) |
5 mg |
333% |
| Vitamin B3 (Niacin) |
10 mg |
50% |
| Vitamin B-6 (Pyridoxine HCL) |
10 mg |
500% |
| Calcium Carbonate (40% Ca) |
50 mg |
2% |
| Sesame Seed Oil (100% Concentration) |
1,398 mg |
† |
| Adipo-X™ Proprietary Blend |
771 mg |
† |
Caffeine Anhydrous USP
L-Tyrosine AKG
Raspberry Ketones 99%
Advantra Z® (Citrus Aurantium 30%)
Capsaicin Extract (Cayenne)
Yohimbe PE (8% Yohimbine)
Paprika Extract
Ginger Root Extract (20% Gingerols)
Coleus Forskohli (40% Forskolin)
Green Tea (90% Polyphenols, 45% EGCG)
Vinpocetine 99%
Bioperine® |
* Daily value (DV) are based on a 2000 calorie
diet
† Daily value (DV) not established |
Other Ingredients: Gelatin, Lecithin and Mixed Tocopherols.
Directions: As a dietary supplement, start with a 1 capsule serving to evaluate
tolerance and gradually increase to a full 3 capsule serving. Take one serving with
8 oz of water 30 to 60 minutes prior to breakfast on an empty stomach and an additional
serving in the early afternoon on an empty stomach. Do not exceed 6 capsules in
a 24 hour period. Do not take 6 hours prior to bed because ADIPO-X™ may interfere
with sleep. Read entire label before using. For even better results, stack with
Axis Labs® SesaMAX®.
Store in a cool place. Protect from heat, light and moisture.
WARNING: KEEP OUT OF REACH OF CHILDREN. Not for use by individuals under
the age of 18 or elderly. Consult a physician before using this or any dietary supplement.
Do not use if pregnant, nursing or chronically ill. This product is not intended
for individuals who are at risk of or have been treated for digestive disorders,
high blood pressure, heart disease, thyroid disease, depression or other psychiatric
conditions, renal disease, reoccurring headaches, spasms, has asthma or taking asthma
medication or if your are using a MAO inhibitor.
Manufactured with pride in the USA.
Adipo-X™ is a registered trademark of Axis Labs®.
* These statements have not been evaluated by the FDA. This product is not intended
to diagnose, treat, cure or prevent any disease.
References:
Akiba, T., K. Yaguchi, et al. (2004). "Inhibitory mechanism of caffeine on insulin-stimulated
glucose uptake in adipose cells." Biochem Pharmacol 68(10): 1929-37.
Banderet, L. E. and H. R. Lieberman (1989). "Treatment with tyrosine, a neurotransmitter
precursor, reduces environmental stress in humans." Brain Res Bull 22(4): 759-62.
Belza, A., E. Frandsen, et al. (2007). "Body fat loss achieved by stimulation of
thermogenesis by a combination of bioactive food ingredients: a placebo-controlled,
double-blind 8-week intervention in obese subjects." Int J Obes (Lond) 31(1): 121-30.
Bhardwaj, R. K., H. Glaeser, et al. (2002). "Piperine, a major constituent of black
pepper, inhibits human P-glycoprotein and CYP3A4." J Pharmacol Exp Ther 302(2):
645-50.
Borea, P. A., K. Varani, et al. (1994). "Full and partial agonistic behaviour and
thermodynamic binding parameters of adenosine A1 receptor ligands." Eur J Pharmacol
267(1): 55-61.
Carlson, L. A., R. W. Butcher, et al. (1970). "Fat mobilizing lipolysis and levels
of cyclic AMP in human and dog adipose tissue." Acta Med Scand 187(6): 525-8.
Chanadiri, T., T. Sanikidze, et al. (2005). "[Effectiveness of green tea catechines
for the correction of the alimentary obesity in the experiment]." Georgian Med News(126):
61-3.
Chaudhry, A. and J. G. Granneman (1991). "Developmental changes in adenylyl cyclase
and GTP binding proteins in brown fat." Am J Physiol 261(2 Pt 2): R403-11.
Chaudhry, A. and J. G. Granneman (1997). "Effect of hypothyroidism on adenylyl cyclase
activity and subtype gene expression in brown adipose tissue." Am J Physiol 273(2
Pt 2): R762-7.
Chen, D., C. Y. Wang, et al. (2005). "Inhibition of human liver catechol-O-methyltransferase
by tea catechins and their metabolites: structure-activity relationship and molecular-modeling
studies." Biochem Pharmacol 69(10): 1523-31.
Choi, H. S. (2006). "Lipolytic effects of citrus peel oils and their components."
J Agric Food Chem 54(9): 3254-8.
Cui, J. and J. Himms-Hagen (1992). "Long-term decrease in body fat and in brown
adipose tissue in capsaicin-desensitized rats." Am J Physiol 262(4 Pt 2): R568-73.
Currie, P. J. and L. M. Wilson (1992). "Yohimbine attenuates clonidine-induced feeding
and macronutrient selection in genetically obese (ob/ob) mice." Pharmacol Biochem
Behav 43(4): 1039-46.
Diepvens, K., E. M. Kovacs, et al. (2005). "Effect of green tea on resting energy
expenditure and substrate oxidation during weight loss in overweight females." Br
J Nutr 94(6): 1026-34.
Diepvens, K., K. R. Westerterp, et al. (2007). "Obesity and thermogenesis related
to the consumption of caffeine, ephedrine, capsaicin, and green tea." Am J Physiol
Regul Integr Comp Physiol 292(1): R77-85.
Dolgacheva, L. P., B. B. Abzhalelov, et al. (2003). "Norepinephrine induces slow
calcium signalling in murine brown preadipocytes through the beta-adrenoceptor/cAMP/protein
kinase A pathway." Cell Signal 15(2): 209-16.
Dulloo, A. G., C. Duret, et al. (1999). "Efficacy of a green tea extract rich in
catechin polyphenols and caffeine in increasing 24-h energy expenditure and fat
oxidation in humans." Am J Clin Nutr 70(6): 1040-5.
Dulloo, A. G., J. Seydoux, et al. (2000). "Green tea and thermogenesis: interactions
between catechin-polyphenols, caffeine and sympathetic activity." Int J Obes Relat
Metab Disord 24(2): 252-8.
Galitzky, J., M. Lafontan, et al. (1993). "Role of vascular alpha-2 adrenoceptors
in regulating lipid mobilization from human adipose tissue." J Clin Invest 91(5):
1997-2003.
Galitzky, J., D. Riviere, et al. (1990). "Pharmacodynamic effects of chronic yohimbine
treatment in healthy volunteers." Eur J Clin Pharmacol 39(5): 447-51.
Galitzky, J., M. Taouis, et al. (1988). "Alpha 2-antagonist compounds and lipid
mobilization: evidence for a lipid mobilizing effect of oral yohimbine in healthy
male volunteers." Eur J Clin Invest 18(6): 587-94.
Gelenberg, A. J., C. J. Gibson, et al. (1982). "Neurotransmitter precursors for
the treatment of depression." Psychopharmacol Bull 18(1): 7-18.
Gomez-Ambrosi, J., G. Fruhbeck, et al. (2001). "Divergent effects of an alpha2-adrenergic
antagonist on lipolysis and thermogenesis: interactions with a beta3-adrenergic
agonist in rats." Int J Mol Med 8(1): 103-9.
Grunfeld, C. and J. K. Shigenaga (1984). "Nicotinamide and other inhibitors of ADP-ribosylation
block deoxyglucose uptake in cultured cells." Biochem Biophys Res Commun 123(2):
785-91.
Han, L. K., X. J. Gong, et al. (2005). "[Antiobesity actions of Zingiber officinale
Roscoe]." Yakugaku Zasshi 125(2): 213-7.
Harmer, C. J., S. F. McTavish, et al. (2001). "Tyrosine depletion attenuates dopamine
function in healthy volunteers." Psychopharmacology (Berl) 154(1): 105-11.
Hellstrom, L., S. Rossner, et al. (1997). "Lipolytic catecholamine resistance linked
to alpha 2-adrenoceptor sensitivity--a metabolic predictor of weight loss in obese
subjects." Int J Obes Relat Metab Disord 21(4): 314-20.
Ho, R. and Q. H. Shi (1982). "Forskolin as a novel lipolytic agent." Biochem Biophys
Res Commun 107(1): 157-64.
Ho, R. J. (1970). "Dependence of hormone-stimulated lipolysis on ATP and cyclic
AMP levels in fat cells." Horm Metab Res 2: Suppl 2:83-7.
Honnor, R. C., G. S. Dhillon, et al. (1985). "cAMP-dependent protein kinase and
lipolysis in rat adipocytes. II. Definition of steady-state relationship with lipolytic
and antilipolytic modulators." J Biol Chem 260(28): 15130-8.
Howald, H. and J. Decombaz (1983). "Nutrient intake and energy regulation in physical
exercise." Experientia Suppl 44: 77-88.
Imbeault, P., D. Prud'Homme, et al. (2000). "Adipose tissue metabolism in young
and middle-aged men after control for total body fatness." J Clin Endocrinol Metab
85(7): 2455-62.
Imbeault, P., A. Tremblay, et al. (2000). "beta-adrenoceptor-stimulated lipolysis
of subcutaneous abdominal adipocytes as a determinant of fat oxidation in obese
men." Eur J Clin Invest 30(4): 290-6.
Kawada, T., K. Hagihara, et al. (1986). "Effects of capsaicin on lipid metabolism
in rats fed a high fat diet." J Nutr 116(7): 1272-8.
Kawada, T., T. Watanabe, et al. (1986). "Capsaicin-induced beta-adrenergic action
on energy metabolism in rats: influence of capsaicin on oxygen consumption, the
respiratory quotient, and substrate utilization." Proc Soc Exp Biol Med 183(2):
250-6.
Kirchgessner, M., D. A. Roth-Maier, et al. (1995). "[B-vitamins (thiamine, vitamin
b6, pantothenic acid) in lean muscle tissue of growing cattle of the German Simmental
breed under different feeding intensities]." Z Lebensm Unters Forsch 201(1): 20-4.
Kobayashi, A., T. Osaka, et al. (1998). "Capsaicin activates heat loss and heat
production simultaneously and independently in rats." Am J Physiol 275(1 Pt 2):
R92-8.
Kucio, C., K. Jonderko, et al. (1991). "Does yohimbine act as a slimming drug?"
Isr J Med Sci 27(10): 550-6.
Lambert, J. D., J. Hong, et al. (2004). "Piperine enhances the bioavailability of
the tea polyphenol (-)-epigallocatechin-3-gallate in mice." J Nutr 134(8): 1948-52.
Le Corre, P., G. Dollo, et al. (1999). "Biopharmaceutics and metabolism of yohimbine
in humans." Eur J Pharm Sci 9(1): 79-84.
Lejeune, M. P., E. M. Kovacs, et al. (2003). "Effect of capsaicin on substrate oxidation
and weight maintenance after modest body-weight loss in human subjects." Br J Nutr
90(3): 651-59.
Lemonnier, D., P. de Gasquet, et al. (1974). "Effect of dietary vitamin B level
on fat storage, adipose tissue cellularity and energy expenditure in rats and mice
fed a high-fat diet." Nutr Metab 16(1): 15-29.
Lieberman, H. R., J. H. Georgelis, et al. (2005). "Tyrosine prevents effects of
hyperthermia on behavior and increases norepinephrine." Physiol Behav 84(1): 33-8.
Lin, Z., Y. Liao, et al. (2007). "Amides from Piper nigrum L. with dissimilar effects
on melanocyte proliferation in-vitro." J Pharm Pharmacol 59(4): 529-36.
Litosch, I., T. H. Hudson, et al. (1982). "Forskolin as an activator of cyclic AMP
accumulation and lipolysis in rat adipocytes." Mol Pharmacol 22(1): 109-15.
Lu, H., X. Meng, et al. (2003). "Enzymology of methylation of tea catechins and
inhibition of catechol-O-methyltransferase by (-)-epigallocatechin gallate." Drug
Metab Dispos 31(5): 572-9.
Malbon, C. C. and M. P. Graziano (1983). "Adenosine deaminase normalizes cyclic
AMP responses of hypothyroid rat fat cells to forskolin, but not beta-adrenergic
agonists." FEBS Lett 155(1): 35-8.
Masuda, Y., S. Haramizu, et al. (2003). "Upregulation of uncoupling proteins by
oral administration of capsiate, a nonpungent capsaicin analog." J Appl Physiol
95(6): 2408-15.
McNamara, F. N., A. Randall, et al. (2005). "Effects of piperine, the pungent component
of black pepper, at the human vanilloid receptor (TRPV1)." Br J Pharmacol 144(6):
781-90.
Melnyk, A. and J. Himms-Hagen (1995). "Resistance to aging-associated obesity in
capsaicin-desensitized rats one year after treatment." Obes Res 3(4): 337-44.
Mochizuki, M. and N. Hasegawa (2004). "Effects of green tea catechin-induced lipolysis
on cytosol glycerol content in differentiated 3T3-L1 cells." Phytother Res 18(11):
945-6.
Morimoto, C., Y. Satoh, et al. (2005). "Anti-obese action of raspberry ketone."
Life Sci 77(2): 194-204.
Muroyama, K., S. Murosaki, et al. (2003). "Effects of intake of a mixture of thiamin,
arginine, caffeine, and citric acid on adiposity in healthy subjects with high percent
body fat." Biosci Biotechnol Biochem 67(11): 2325-33.
Muroyama, K., S. Murosaki, et al. (2003). "Anti-obesity effects of a mixture of
thiamin, arginine, caffeine, and citric acid in non-insulin dependent diabetic KK
mice." J Nutr Sci Vitaminol (Tokyo) 49(1): 56-63.
Ohnuki, K., S. Haramizu, et al. (2001). "Administration of capsiate, a non-pungent
capsaicin analog, promotes energy metabolism and suppresses body fat accumulation
in mice." Biosci Biotechnol Biochem 65(12): 2735-40.
Ostman, J. (1964). "Effect Of Nicotinic Acid On The Fatty Acid Metabolism Of Adipose
Tissue In Alloxan Diabetic Rats." Metabolism 13: 675-80.
Ostman, J. (1965). "Inhibitory Effect Of Nicotinic Acid On Fat Mobilization In Alloxan-Diabetic
Rats. Ii. A Comparison Of The Effect Of Nicotinic Acid And Salicylate On The Fatty
Acid Metabolism And Glucose Uptake By Adipose Tissue In Vitro." Acta Med Scand 177:
623-9.
Papadelis, C., C. Kourtidou-Papadeli, et al. (2003). "Effects of mental workload
and caffeine on catecholamines and blood pressure compared to performance variations."
Brain Cogn 51(1): 143-54.
Rababah, T. M., N. S. Hettiarachchy, et al. (2004). "Total phenolics and antioxidant
activities of fenugreek, green tea, black tea, grape seed, ginger, rosemary, gotu
kola, and ginkgo extracts, vitamin E, and tert-butylhydroquinone." J Agric Food
Chem 52(16): 5183-6.
Razzoli, G. (1972). "[Effect of coffee on deposit lipids]." Minerva Med 63(61):
3361-2.
Roberts, S. A., J. M. Thorpe, et al. (2001). "Tyrosine requirement of healthy men
receiving a fixed phenylalanine intake determined by using indicator amino acid
oxidation." Am J Clin Nutr 73(2): 276-82.
Scarpace, P. J., L. A. Baresi, et al. (1987). "Modulation of receptors and adenylate
cyclase activity during sucrose feeding, food deprivation, and cold exposure." Am
J Physiol 253(6 Pt 1): E629-35.
Schimmel, R. J. (1980). "Interactions between catecholamines, methyl xanthines and
adenosine in regulation of cyclic AMP accumulation in hamster adipocytes." Biochim
Biophys Acta 629(1): 83-94.
Shixian, Q., B. VanCrey, et al. (2006). "Green tea extract thermogenesis-induced
weight loss by epigallocatechin gallate inhibition of catechol-O-methyltransferase."
J Med Food 9(4): 451-8.
Simon, E., M. T. Macarulla, et al. (2005). "Body fat-lowering effect of conjugated
linoleic acid is not due to increased lipolysis." J Physiol Biochem 61(2): 363-9.
Srinivasan, M. R. and M. N. Satyanarayana (1989). "Effect of capsaicin on skeletal
muscle lipoprotein lipase in rats fed high fat diet." Indian J Exp Biol 27(10):
910-2.
Szallasi, A. (2005). "Piperine: researchers discover new flavor in an ancient spice."
Trends Pharmacol Sci 26(9): 437-9.
Szilagyi, G., Z. Nagy, et al. (2005). "Effects of vinpocetine on the redistribution
of cerebral blood flow and glucose metabolism in chronic ischemic stroke patients:
a PET study." J Neurol Sci 229-230: 275-84.
Tam, S. W., M. Worcel, et al. (2001). "Yohimbine: a clinical review." Pharmacol
Ther 91(3): 215-43.
Tapsell, L. C., I. Hemphill, et al. (2006). "Health benefits of herbs and spices:
the past, the present, the future." Med J Aust 185(4 Suppl): S4-24.
Tornqvist, H., L. Krabisch, et al. (1972). "Rapid assay for hormone-sensitive lipase
activity of adipose tissue." J Lipid Res 13(3): 424-6.
Tsi, D., A. K. Nah, et al. (2003). "Clinical study on the combined effect of capsaicin,
green tea extract and essence of chicken on body fat content in human subjects."
J Nutr Sci Vitaminol (Tokyo) 49(6): 437-41.
Verma, S. K. and A. Bordia (2001). "Ginger, fat and fibrinolysis." Indian J Med
Sci 55(2): 83-6.
Walldius, G. and G. Wahlberg (1985). "Effects of nicotinic acid and its derivatives
on lipid metabolism and other metabolic factors related to atherosclerosis." Adv
Exp Med Biol 183: 281-93.
Westerterp-Plantenga, M., K. Diepvens, et al. (2006). "Metabolic effects of spices,
teas, and caffeine." Physiol Behav 89(1): 85-91.
Wolfram, S., Y. Wang, et al. (2006). "Anti-obesity effects of green tea: from bedside
to bench." Mol Nutr Food Res 50(2): 176-87.
Wu, A. H., C. C. Tseng, et al. (2003). "Tea intake, COMT genotype, and breast cancer
in Asian-American women." Cancer Res 63(21): 7526-9.
Yoshida, T., K. Yoshioka, et al. (1988). "Effects of capsaicin and isothiocyanate
on thermogenesis of interscapular brown adipose tissue in rats." J Nutr Sci Vitaminol
(Tokyo) 34(6): 587-94.
Zhang, L. L., D. Yan Liu, et al. (2007). "Activation of transient receptor potential
vanilloid type-1 channel prevents adipogenesis and obesity." Circ Res 100(7): 1063-70.
